DECA-VX

DECA-VX is unique (so far as I know) in that 5a -reductase, the
enzyme which converts testosterone to the more-potent DHT,
actually converts nandrolone to a less-potent compound.
Therefore this AAS is somewhat deactivated in the skin, scalp,
and prostate, and these tissues experience an effectively-lower
androgen level than the rest of the body. Therefore, for the
same amount of activity as another drug at the androgen
receptors (ARs) in muscle tissue, Deca gives less activity in the
scalp, skin, and prostate. Thus, it is the best choice for those
particularly concerned with these things. Its effectiveness at the
androgen receptor of muscle tissue is superior to that of
testosterone: it binds better. Yet, it gives only about half the
muscle-building results per milligram. This I think is a result of
its being less effective or entirely ineffective in
non-AR-mediated mechanisms for muscle growth. It also
appears less effective or entirely ineffective in activity on nerve
cells, certainly on the nerve cells responsible for erectile
function. Use of Deca as the sole AAS often results in complete
inability to perform sexually. These problems can be solved by
combining with a drug that does supply the missing activity:
e.g. testosterone. Nandrolone is proven to be a progestin. This
fact is of clear importance in bodybuilding, because while
moderate Deca-only use actually lowers estrogen levels as a
consequence of reducing natural testosterone levels and thus
allowing the aromatase enzyme less substrate to work with,
Deca nonetheless can cause gyno in some individuals.
Furthermore, just as progesterone will to a point increase sex
drive in women, and then often decrease it as levels get too
high, high levels of progestogenic steroids can kill sex drive in
male bodybuilders, though there is a great deal of individual
variability as to what is too much. Incidentally, this
progestogenic activity also inhibits LH production, and contrary
to common belief, even small amounts of Deca are quite
inhibitory, approximately as much so as the same amount of
testosterone. To some extent, nandrolone aromatizes to
estrogen, and it does not appear that this can be entirely
blocked by use of aromatase inhibitors - indeed, aromatase
may not be involved at all in this process (there is no evidence
in humans that such occurs) with the enzyme CYP 2C11 being
in my opinion the more likely candidate for this activity. In any
case, Cytadren, an aromatase inhibitor, has not been found
effective in avoiding aromatization of nandrolone.

Nandrolone deconate
10ml in Vial 250mg per one ml
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